Tom Haviland joins me for the SEVENTEENTH interview in an ongoing series of interviews in which people from around the world express their views regarding the mRNA injections.
path name timeframe initiating_event mechanistic_steps key_drivers detectable_markers mitigation
P1 Heparin-displacement and ternary complexes minutes–hours mRNA → local Spike expression Injected heparin competes with cell-surface HS; Spike is displaced into plasma as Spike–heparin complexes; ternary complexes form with fibrinogen or PF4 altering coagulation Heparin chain length and dose; Spike local concentration; presence of fibrinogen, PF4, cations Transient rise in soluble Spike antigen; Spike co‑immunoprecipitated with heparin; altered thrombin generation Prefer LMWH when appropriate; monitor platelet counts and PF4 antibodies; anticoagulation management
P2 Spike-induced amyloid-like fibrin hours–days mRNA → Spike present during coagulation events Spike interacts with fibrinogen/fibrin promoting abnormal amyloid-like polymerization; fibrin becomes resistant to plasmin and persists as microclots Spike concentration at clot sites; cofactors promoting amyloidogenesis; host fibrinolytic capacity Fibrin resistant to fibrinolysis; dense atypical fibers on EM; elevated D-dimer with poor clot resolution Enhance fibrinolysis in severe cases; investigate amyloid-disrupting agents; monitor microvascular function
P3 Protein–polyanion coacervates and scaffolded coagulation minutes–days high local Spike + long polyanions present Spike plus long anionic polymers (HS, polyP, extracellular nucleic acids) and multivalent cations drive LLPS or precipitation; condensates concentrate coagulation factors and platelets, lowering thrombin threshold Polyanion chain length and concentration; Ca2+/Mg2+; multivalent protein partners (fibrinogen, histones) Microscopy showing droplets/particles; turbidity/DLS changes; enrichment of coagulation factors in particulate fractions Enzymatic degradation of polyanions experimentally; anticoagulation to prevent condensate-driven thrombin bursts
P4 Immune complex / PF4-mediated platelet activation days antibody formation or PF4 release after platelet activation PF4–polyanion complexes become immunogenic; anti-PF4 or crosslinking antibodies trigger FcγR-mediated platelet activation; amplified PF4 release and NETosis produce systemic thrombosis Timing of antibody formation; prior sensitization; platelet activation level Thrombocytopenia with thrombosis; positive PF4–heparin ELISA; platelet activation assays Avoid heparin if immune PF4 complexes suspected; use non-heparin anticoagulants; immunomodulation (IVIG)
P5 Endothelial activation and complement amplification hours–days Spike exposure to endothelium or endothelial uptake Spike induces endothelial activation with tissue factor and vWF release; complement activation amplifies inflammation and coagulation; microthrombi form and can propagate systemically Endothelial exposure to Spike; baseline endothelial health; complement regulation Elevated vWF and soluble thrombomodulin; complement split products (C3a, C5a); microthrombi on histology Target endothelial inflammation and complement; anticoagulation alongside anti-inflammatory therapy
P6 NETosis and histone-mediated thrombosis hours–days local inflammation triggers neutrophil activation Neutrophils release NETs rich in DNA and histones; NETs scaffold platelets and coagulation factors promoting thrombosis Neutrophil activation; PAD4 activity; extracellular DNA; histones Cell-free DNA; citrullinated histone H3; platelet–NET aggregates; elevated thrombin generation DNase treatment in vitro; anti-histone antibodies; anticoagulation; anti-inflammatory therapy
P7 Platelet microparticle seeding minutes–hours platelet activation at injection site or systemically Activated platelets shed microparticles exposing phosphatidylserine that nucleate thrombin and bind fibrinogen; microparticles circulate and seed distal clotting Platelet activation; shear stress; thrombin; exposed PS Elevated circulating platelet microparticles; annexin V+ particles; increased procoagulant activity Antiplatelet agents; anticoagulation; reduce platelet activation triggers
P8 Extracellular vesicle (EV) transport of Spike hours–days cells package Spike into EVs and release them Spike-bearing EVs circulate and present multivalent Spike on vesicle surface, binding polyanions and recruiting coagulation factors EV biogenesis; membrane display of Spike; opsonization Spike detected in EV fractions; nanoparticle tracking; EV proteomics showing Spike and coagulation factors EV isolation and characterization; immunocapture; anticoagulation if procoagulant
P10 Protease-mediated fibrin remodeling and resistant fragments hours–days local protease release (neutrophil elastase, bacterial proteases) Proteases partially degrade fibrin producing atypical fragments that resist fibrinolysis and seed new clot formation Protease release; altered plasmin activity; crosslinking by FXIII Abnormal fibrin degradation products; altered D-dimer patterns; plasmin resistance in vitro Protease inhibitors; enhance plasmin activity; targeted fibrinolysis
P11 Lipid nanoparticle (LNP)-protein aggregation minutes–hours LNPs carrying mRNA interact with extracellular proteins LNPs bind serum proteins including fibrinogen or complement components, forming a protein corona that can be procoagulant or immunogenic LNP surface chemistry; protein corona composition; charge interactions Protein corona profiling; co-isolation of LNPs with coagulation factors; complement activation Modify LNP composition; monitor protein corona; anticoagulation if indicated
P12 Antibody-mediated crosslinking and immune complexes days–weeks anti-Spike antibodies form after vaccination Antibodies crosslink Spike or Spike-containing fragments creating large immune complexes that activate complement and FcγR on platelets/leukocytes Timing of seroconversion; antibody isotype and affinity; Fc receptor engagement Circulating immune complexes; complement activation; platelet activation assays Immunomodulation (IVIG); avoid heparin if PF4 involvement suspected; non-heparin anticoagulants
P13 Bacterial translocation or endotoxin-triggered coagulation hours–days local tissue disruption allows bacterial products into circulation LPS and other PAMPs trigger systemic inflammation, endothelial activation, tissue factor expression and disseminated coagulation Gut or tissue barrier breach; innate immune activation; TLR signaling Elevated LPS; cytokines (IL-6); tissue factor; D-dimer Treat infection; anti-inflammatory therapy; supportive anticoagulation
P14 Oxidative stress–induced endothelial damage hours–days local oxidative burst or systemic ROS increase ROS damage endothelial glycocalyx and cells exposing subendothelial procoagulant surfaces and releasing vWF and TF Oxidative stress; reduced NO; glycocalyx shedding Elevated soluble thrombomodulin; vWF; markers of oxidative stress Antioxidants experimentally; endothelial protective strategies; anticoagulation
P15 Protein misfolding,heterologous amyloid seeding (non-fibrin) days Spike or vaccine components seed misfolding of host proteins Misfolded proteins form amyloid-like aggregates that bind coagulation factors or platelets, altering hemostasis Amyloidogenic sequences; molecular chaperone overload; proteostasis stress Amyloid staining of plasma particulates; protease-resistant aggregates; altered clot structure Chaperone modulators experimentally; enhance clearance pathways; anticoagulation if prothrombotic
P16 Metal-mediated precipitation, mixed inorganic–protein particles minutes–hours local release or presence of polyvalent metals (Fe, Ca, Mn) Metals bridge polyanions and proteins forming mixed inorganic–protein precipitates that trap coagulation factors and cells Metal availability; pH; polyanion concentration Particulate metal content by ICP-MS; co-precipitation of coagulation proteins; microscopy Chelation in vitro; control metal exposure; anticoagulation
Here is a table of Clotting Pathways that are possible from the mRNA novel coronavirus vaccines.
I think injecting heparin is a bad idea if something like VITT is going on.This table lists non-amyloid clots as well - amyloidosis may or may not be happening. Sufficent but not necessary... lots of potential parallel clotting action in this table.
P1 Heparin-displacement and ternary complexes
TIMEFRAME minutes–hours
INITIATING EVENT mRNA → local Spike expression
MECHANISTIC STEPS Injected heparin competes with cell-surface HS; Spike is displaced into plasma as Spike–heparin complexes; ternary complexes form with fibrinogen or PF4 altering coagulation
KEY DRIVERS Heparin chain length and dose; Spike local concentration; presence of fibrinogen, PF4, cations
DETECTABLE MARKERS Transient rise in soluble Spike antigen; Spike co‑immunoprecipitated with heparin; altered thrombin generation
MITIGATION Prefer LMWH when appropriate; monitor platelet counts and PF4 antibodies; anticoagulation management
P2 Spike-induced amyloid-like fibrin
TIMEFRAME hours–days
INITIATING EVENT mRNA → Spike present during coagulation events
MECHANISTIC STEPS Spike interacts with fibrinogen/fibrin promoting abnormal amyloid-like polymerization; fibrin becomes resistant to plasmin and persists as microclots
MITIGATION Avoid heparin if immune PF4 complexes suspected; use non-heparin anticoagulants; immunomodulation (IVIG)
P5 Endothelial activation and complement amplification
TIMEFRAME hours–days
INITIATING EVENT Spike exposure to endothelium or endothelial uptake
MECHANISTIC STEPS Spike induces endothelial activation with tissue factor and vWF release; complement activation amplifies inflammation and coagulation; microthrombi form and can propagate systemically
INITIATING EVENT LNPs carrying mRNA interact with extracellular proteins
MECHANISTIC STEPS LNPs bind serum proteins including fibrinogen or complement components, forming a protein corona that can be procoagulant or immunogenic
KEY DRIVERS LNP surface chemistry; protein corona composition; charge interactions
DETECTABLE MARKERS Protein corona profiling; co-isolation of LNPs with coagulation factors; complement activation
MITIGATION Modify LNP composition; monitor protein corona; anticoagulation if indicated
P12 Antibody-mediated crosslinking and immune complexes
TIMEFRAME days–weeks
INITIATING EVENT anti-Spike antibodies form after vaccination
MECHANISTIC STEPS Antibodies crosslink Spike or Spike-containing fragments creating large immune complexes that activate complement and FcγR on platelets/leukocytes
KEY DRIVERS Timing of seroconversion; antibody isotype and affinity; Fc receptor engagement
Introduction: I write this as an educator and researcher who has spent years observing how large systems fracture human bonds while presenting themselves as benevolent guardians. When those systems fail—when they harm rather than heal—the damage is not merely statistical. It is personal, intimate, and enduring. Lives are lost, families are shattered, and grief is often driven underground by silence, ridicule, or fear. What follows is an educational presentation rooted in a simple but profound idea: that ordinary people, acting locally and peacefully, can create meaning, solidarity, and resistance through ritual. This essay proposes a recurring, silent act of remembrance for those believed to have died as a result of the mRNA injections, with good reason and explores why such a ritual matters—not only for mourning, but for community formation, moral clarity, and future resilience.
It's like talking to your cat or dog about the mRNA vax/jab they know you care and only mean them well esp at feeding time,
Only our Pets have an excuse = they're animals:
ALL this info I sent revealing every immune boosting food, supplement, herb & drink for optimum health as soon as Covid was announced was coming in Fed., 2020- and I never get a cold or flu,
I then explained all these helps WITH dozensof links so they could validate the scientific research = all so they won't need to get the mRNA Vax when it comes out in 2021.
Sent out to these folks every fact including that *every test animal died given that Vax, and that it was NOT properly vetted after that but WAS just allowed by the CDC+FDA,
And that POTUS Trump was lied to about its effectiveness and reshared that Trump also 1st gave *all the natural immune boosters sent to him by the great Dr. Zelenko,
And also that Fauci was a lying monster about AIDS medicine and 10's of thousands died because of it, and theorphaned black children in NYC he experimented on and killed,
Andthe many Beagle dogs he tortured to death cutting their voice boxs so not to hear the screams,
Still even now most of these same people I reached out to all along do not seem to feel there's a problem,
I have sent this Tom Haviland article by Jim Roguski here to them all already twiceand it'll lijely be ignored:
The viscosity data here is especially compelling because its such a measurable physical property. Going from 30% to 40% grape jelly clots while also seeing these new fibrous structures isn't just anecdotal - embalmers are basically running an unintentional longitudinal study with huge sample sizes. The fact that this pattern held steady across three years of surveys (27-30% prevalence) suggests whatever mechanism is causing this reached some kind of equilibrium. The silence from regulatory agencies tells you everythingyou need to know about institutional priorities.
This is awful. No one is being told the truth. Did the deceased die from the clots, or from something else, and are the clots being found during embalming?
Almost always the death certificate just says stroke or "myocardial infarction" (i.e., heart attack) or generic "thrombosis" without describing the TYPE of clot that caused the stroke or heart attack. Very disingenuous if you ask me. These death certificates should be much more specific. I think that they are hiding the fact that many of these deaths are being caused by the WHITE FIBROUS CLOTS on purpose.
Thank you so much for interviewing Tom Haviland. More people should be made aware of these clots. I follow Laura and Tom on Substack, after I heard her speak on a local radio show a year ago.
As a Christian why do you have them sware to speak truth it's against the one your swearing upon. Yes is to be yes and no to be no. It against the one your swearing upon. Wrong is wrong.
That was an excellent interview which covered all the important points.
I have repeatedly written to my government (in the UK) and had no responses - from my MP or from individual government departments. Letters sent registered mail are not recorded as received. Apparently all mail into the UK government is first "screened" by security so I cannot even know whether this correspondence reached the right desk.
Discussion or questioning of the covid injections is clearly a classified security matter.
It was a "signed for" letter - after no response from e-mails.
It was registered mail and tracked OK in the post until it got to Westminster - then nothing!
Registered mail should get a signature to prove receipt. Clearly discussion of the pandemic or the injections goes to security and is classified, so they will not sign or acknowledge receipt..
Thank you James for discussing the embalmer clots . The actual embalmers and their assistants need to be aware of the survey, not just the directors. This topic needs to go viral on X and Instagram and other media platforms. We the People must get the word out . Thanks again to Tom and Laura for their efforts to tell the truth . Repeal the Prep act too . 🙏
"Sally" noticed that the death rates in Pilipino CHILDREN rose when the ADULT vaccines rolled out. Shedding?
The fertility rates have dropped substantially ion all countries she follows. It may be that some of the statistical effects suggest targeting of certain populations for specific results, but it is too early yet to know, and preexisting population trends complicate the picture.
Can't say this often enough! KEEP FIGHTING! All the perps who pushed this greatest crime against humanity, all the way down to the local level, must get their comeuppances!
MISTAKES! WERE NOT MADE!
It was NEVER about health! The Powers That Should Not Be were ALWAYS about they want you DEAD or a SLAVE! This is a painful truth to accept but we the people must wake up and fight back! And toxic injections/pills were/are a huge part of their arsenal!
Can't say this often enough! The Military/Industrial Complex and the Biowarfare/industrial Complex, WEF agenda and the evils assaulting humanity are from one and the same source - it is the 99% against the diabolical GREED of the 0.01% who should not be in charge of anything!
The monsters in human skin suits who rule the world get a sadistic vampiric thrill and boost from perpetrating the vilest most demonic crimes against the most vulnerable (babies and small children) and then corrupting the system to get away with it scot free! We the People must stop them, there are a lot more of us than them!
And BIG pHARMa is an arsenal making permanently sickly addicted slaves dependent on their products - the complete opposite of actual health.
Peddling pure poison! Folks have to wake up to reality: health comes from organic diet, daily exercise and clean living and never from a needle or a pill except in dire, rare traumatic injuries.
Can't say this often enough!
SCREW THE HYPOCHONDRIA GERMAPHOBIC FEAR HYSTERIA! DO NOT CONSENT! Avian flu is for the birds! RESIST!
Proudly ANTI-VAXX! Reiterating for the sake of newbies and to support this post.
Ban all vaccine jabs! There has never been a 'safe and effective' vaccine since Edward Jenner's fraud over 200 years ago as per 'Dissolving Illusions' by Suzanne Humphries and 'Turtles All the Way Down' by Anonymous. Health can never come from a needle or pills, but from healthy eating, healthy exercise and healthy living! virustruth.net
JAB INJURIES: GROSS CALAMARI BLOOD CLOTS/AUTISM TSUNAMI/SADS/TURBO CANCER/BIZARRE TERMINAL ILLNESSES: More tragic victims of the ruling parasite genocidal enslavement agenda, sacrificed on the altar of psychopathic greed and hatred of humanity.
Divide and rule! Agents provocateurs anyone, FALSE FLAGS, propaganda social engineering psyops? Keeping us proles at each others' throats while the globalist technocrat predators laugh all the way to the BIS and The Bank of Rothschild's!
BURN BACK BETTER!
HELL NO TO GENESIS MISSION/STARGATE/DEEPSEEK! HELL NO TO AI! technocracy.news
Life everywhere is being assaulted by THE TECHNOCRATIC OMNIWAR! RESIST! DO NOT CONSENT TO ALL THINGS DIGITAL, 'SMART', AI, 5G, NO CASH - ALL OF IT! dhughes.substack.com Technocrat ruling class psychos get a sadistic thrill from their powers over life and death and hurting all who stand in their way and they need the resources worldwide to build their digital total slavery control grids (herd survivors into 15 minute city digital prisons)!
AI is designed to be anti-human/anti-life programmed by technocrat control freak psychos - garbage in = garbage out. Everyone got along just fine without all these absurd and downright satanic electronic gadgets that did not exist until recently. NOBODY NEEDS THIS AI CRAP!
PSYCHOPATHS! MEGALOMANIACS!
This horrifying Congress Critters, Gates, Governor 'Gruesome Newscum', 'Lone Scum', Soros, 'Benedict' Biden and Harris and even Trump, Vance, and 'Ramaswampy' et al are blatant fully owned and operated puppets of their globalist technocrat parasite masters same as other numerous 'PUBLIC SERPENTS' infesting by design from above, the bureaucratic apparatus.
CREATIVITY! ARTISTRY! IMAGINATION! SPIRITUALITY! HUMOR! LOVING KINDNESS! These are the best ways to fight THEM!
I was at emergency the other day in Pembroke, On. Canada and the triage nurse adk if I had
the flu and covid jabs I told her no to both and further said I would not ever take an MRNA jab! I was shocked they are still pushing this not safe and not effective jab! I believe the flu
shot is now MRNA! Last flu shot 2019 gave me a slight stroke 5 days after! I now do not
Great, important interview. James, your NotSafeandNotEffective website is the best and most thorough compilation of covid shot information I have seen, so I share it as much as possible. Thank you for this greate site.
So much death n destruction to come to terms with. Even more demanding- to come to terms with , the complicity of our governments and medical professionals .
path name timeframe initiating_event mechanistic_steps key_drivers detectable_markers mitigation
P1 Heparin-displacement and ternary complexes minutes–hours mRNA → local Spike expression Injected heparin competes with cell-surface HS; Spike is displaced into plasma as Spike–heparin complexes; ternary complexes form with fibrinogen or PF4 altering coagulation Heparin chain length and dose; Spike local concentration; presence of fibrinogen, PF4, cations Transient rise in soluble Spike antigen; Spike co‑immunoprecipitated with heparin; altered thrombin generation Prefer LMWH when appropriate; monitor platelet counts and PF4 antibodies; anticoagulation management
P2 Spike-induced amyloid-like fibrin hours–days mRNA → Spike present during coagulation events Spike interacts with fibrinogen/fibrin promoting abnormal amyloid-like polymerization; fibrin becomes resistant to plasmin and persists as microclots Spike concentration at clot sites; cofactors promoting amyloidogenesis; host fibrinolytic capacity Fibrin resistant to fibrinolysis; dense atypical fibers on EM; elevated D-dimer with poor clot resolution Enhance fibrinolysis in severe cases; investigate amyloid-disrupting agents; monitor microvascular function
P3 Protein–polyanion coacervates and scaffolded coagulation minutes–days high local Spike + long polyanions present Spike plus long anionic polymers (HS, polyP, extracellular nucleic acids) and multivalent cations drive LLPS or precipitation; condensates concentrate coagulation factors and platelets, lowering thrombin threshold Polyanion chain length and concentration; Ca2+/Mg2+; multivalent protein partners (fibrinogen, histones) Microscopy showing droplets/particles; turbidity/DLS changes; enrichment of coagulation factors in particulate fractions Enzymatic degradation of polyanions experimentally; anticoagulation to prevent condensate-driven thrombin bursts
P4 Immune complex / PF4-mediated platelet activation days antibody formation or PF4 release after platelet activation PF4–polyanion complexes become immunogenic; anti-PF4 or crosslinking antibodies trigger FcγR-mediated platelet activation; amplified PF4 release and NETosis produce systemic thrombosis Timing of antibody formation; prior sensitization; platelet activation level Thrombocytopenia with thrombosis; positive PF4–heparin ELISA; platelet activation assays Avoid heparin if immune PF4 complexes suspected; use non-heparin anticoagulants; immunomodulation (IVIG)
P5 Endothelial activation and complement amplification hours–days Spike exposure to endothelium or endothelial uptake Spike induces endothelial activation with tissue factor and vWF release; complement activation amplifies inflammation and coagulation; microthrombi form and can propagate systemically Endothelial exposure to Spike; baseline endothelial health; complement regulation Elevated vWF and soluble thrombomodulin; complement split products (C3a, C5a); microthrombi on histology Target endothelial inflammation and complement; anticoagulation alongside anti-inflammatory therapy
P6 NETosis and histone-mediated thrombosis hours–days local inflammation triggers neutrophil activation Neutrophils release NETs rich in DNA and histones; NETs scaffold platelets and coagulation factors promoting thrombosis Neutrophil activation; PAD4 activity; extracellular DNA; histones Cell-free DNA; citrullinated histone H3; platelet–NET aggregates; elevated thrombin generation DNase treatment in vitro; anti-histone antibodies; anticoagulation; anti-inflammatory therapy
P7 Platelet microparticle seeding minutes–hours platelet activation at injection site or systemically Activated platelets shed microparticles exposing phosphatidylserine that nucleate thrombin and bind fibrinogen; microparticles circulate and seed distal clotting Platelet activation; shear stress; thrombin; exposed PS Elevated circulating platelet microparticles; annexin V+ particles; increased procoagulant activity Antiplatelet agents; anticoagulation; reduce platelet activation triggers
P8 Extracellular vesicle (EV) transport of Spike hours–days cells package Spike into EVs and release them Spike-bearing EVs circulate and present multivalent Spike on vesicle surface, binding polyanions and recruiting coagulation factors EV biogenesis; membrane display of Spike; opsonization Spike detected in EV fractions; nanoparticle tracking; EV proteomics showing Spike and coagulation factors EV isolation and characterization; immunocapture; anticoagulation if procoagulant
P9 Complement-opsonized aggregate formation hours–days Spike or Spike complexes activate complement Complement deposition opsonizes complexes, promotes endothelial activation and leukocyte recruitment, amplifying coagulation via tissue factor Classical/alternative complement activation; C3b/C5a generation Elevated C3a/C5a; complement fragments on complexes; endothelial activation markers Complement inhibitors; anti-inflammatory therapy; anticoagulation
P10 Protease-mediated fibrin remodeling and resistant fragments hours–days local protease release (neutrophil elastase, bacterial proteases) Proteases partially degrade fibrin producing atypical fragments that resist fibrinolysis and seed new clot formation Protease release; altered plasmin activity; crosslinking by FXIII Abnormal fibrin degradation products; altered D-dimer patterns; plasmin resistance in vitro Protease inhibitors; enhance plasmin activity; targeted fibrinolysis
P11 Lipid nanoparticle (LNP)-protein aggregation minutes–hours LNPs carrying mRNA interact with extracellular proteins LNPs bind serum proteins including fibrinogen or complement components, forming a protein corona that can be procoagulant or immunogenic LNP surface chemistry; protein corona composition; charge interactions Protein corona profiling; co-isolation of LNPs with coagulation factors; complement activation Modify LNP composition; monitor protein corona; anticoagulation if indicated
P12 Antibody-mediated crosslinking and immune complexes days–weeks anti-Spike antibodies form after vaccination Antibodies crosslink Spike or Spike-containing fragments creating large immune complexes that activate complement and FcγR on platelets/leukocytes Timing of seroconversion; antibody isotype and affinity; Fc receptor engagement Circulating immune complexes; complement activation; platelet activation assays Immunomodulation (IVIG); avoid heparin if PF4 involvement suspected; non-heparin anticoagulants
P13 Bacterial translocation or endotoxin-triggered coagulation hours–days local tissue disruption allows bacterial products into circulation LPS and other PAMPs trigger systemic inflammation, endothelial activation, tissue factor expression and disseminated coagulation Gut or tissue barrier breach; innate immune activation; TLR signaling Elevated LPS; cytokines (IL-6); tissue factor; D-dimer Treat infection; anti-inflammatory therapy; supportive anticoagulation
P14 Oxidative stress–induced endothelial damage hours–days local oxidative burst or systemic ROS increase ROS damage endothelial glycocalyx and cells exposing subendothelial procoagulant surfaces and releasing vWF and TF Oxidative stress; reduced NO; glycocalyx shedding Elevated soluble thrombomodulin; vWF; markers of oxidative stress Antioxidants experimentally; endothelial protective strategies; anticoagulation
P15 Protein misfolding,heterologous amyloid seeding (non-fibrin) days Spike or vaccine components seed misfolding of host proteins Misfolded proteins form amyloid-like aggregates that bind coagulation factors or platelets, altering hemostasis Amyloidogenic sequences; molecular chaperone overload; proteostasis stress Amyloid staining of plasma particulates; protease-resistant aggregates; altered clot structure Chaperone modulators experimentally; enhance clearance pathways; anticoagulation if prothrombotic
P16 Metal-mediated precipitation, mixed inorganic–protein particles minutes–hours local release or presence of polyvalent metals (Fe, Ca, Mn) Metals bridge polyanions and proteins forming mixed inorganic–protein precipitates that trap coagulation factors and cells Metal availability; pH; polyanion concentration Particulate metal content by ICP-MS; co-precipitation of coagulation proteins; microscopy Chelation in vitro; control metal exposure; anticoagulation
Here is a table of Clotting Pathways that are possible from the mRNA novel coronavirus vaccines.
I think injecting heparin is a bad idea if something like VITT is going on.This table lists non-amyloid clots as well - amyloidosis may or may not be happening. Sufficent but not necessary... lots of potential parallel clotting action in this table.
P1 Heparin-displacement and ternary complexes
TIMEFRAME minutes–hours
INITIATING EVENT mRNA → local Spike expression
MECHANISTIC STEPS Injected heparin competes with cell-surface HS; Spike is displaced into plasma as Spike–heparin complexes; ternary complexes form with fibrinogen or PF4 altering coagulation
KEY DRIVERS Heparin chain length and dose; Spike local concentration; presence of fibrinogen, PF4, cations
DETECTABLE MARKERS Transient rise in soluble Spike antigen; Spike co‑immunoprecipitated with heparin; altered thrombin generation
MITIGATION Prefer LMWH when appropriate; monitor platelet counts and PF4 antibodies; anticoagulation management
P2 Spike-induced amyloid-like fibrin
TIMEFRAME hours–days
INITIATING EVENT mRNA → Spike present during coagulation events
MECHANISTIC STEPS Spike interacts with fibrinogen/fibrin promoting abnormal amyloid-like polymerization; fibrin becomes resistant to plasmin and persists as microclots
KEY DRIVERS Spike concentration at clot sites; cofactors promoting amyloidogenesis; host fibrinolytic capacity
DETECTABLE MARKERS Fibrin resistant to fibrinolysis; dense atypical fibers on EM; elevated D-dimer with poor clot resolution
MITIGATION Enhance fibrinolysis in severe cases; investigate amyloid-disrupting agents; monitor microvascular function
P3 Protein–polyanion coacervates and scaffolded coagulation
TIMEFRAME minutes–days
INITIATING EVENT high local Spike + long polyanions present
MECHANISTIC STEPS Spike plus long anionic polymers (HS, polyP, extracellular nucleic acids) and multivalent cations drive LLPS or precipitation; condensates concentrate coagulation factors and platelets, lowering thrombin threshold
KEY DRIVERS Polyanion chain length and concentration; Ca2+/Mg2+; multivalent protein partners (fibrinogen, histones)
DETECTABLE MARKERS Microscopy showing droplets/particles; turbidity/DLS changes; enrichment of coagulation factors in particulate fractions
MITIGATION Enzymatic degradation of polyanions experimentally; anticoagulation to prevent condensate-driven thrombin bursts
P4 Immune complex / PF4-mediated platelet activation
TIMEFRAME days
INITIATING EVENT antibody formation or PF4 release after platelet activation
MECHANISTIC STEPS PF4–polyanion complexes become immunogenic; anti-PF4 or crosslinking antibodies trigger FcγR-mediated platelet activation; amplified PF4 release and NETosis produce systemic thrombosis
KEY DRIVERS Timing of antibody formation; prior sensitization; platelet activation level
DETECTABLE MARKERS Thrombocytopenia with thrombosis; positive PF4–heparin ELISA; platelet activation assays
MITIGATION Avoid heparin if immune PF4 complexes suspected; use non-heparin anticoagulants; immunomodulation (IVIG)
P5 Endothelial activation and complement amplification
TIMEFRAME hours–days
INITIATING EVENT Spike exposure to endothelium or endothelial uptake
MECHANISTIC STEPS Spike induces endothelial activation with tissue factor and vWF release; complement activation amplifies inflammation and coagulation; microthrombi form and can propagate systemically
KEY DRIVERS Endothelial exposure to Spike; baseline endothelial health; complement regulation
DETECTABLE MARKERS Elevated vWF and soluble thrombomodulin; complement split products (C3a, C5a); microthrombi on histology
MITIGATION Target endothelial inflammation and complement; anticoagulation alongside anti-inflammatory therapy
P6 NETosis and histone-mediated thrombosis
TIMEFRAME hours–days
INITIATING EVENT local inflammation triggers neutrophil activation
MECHANISTIC STEPS Neutrophils release NETs rich in DNA and histones; NETs scaffold platelets and coagulation factors promoting thrombosis
KEY DRIVERS Neutrophil activation; PAD4 activity; extracellular DNA; histones
DETECTABLE MARKERS Cell-free DNA; citrullinated histone H3; platelet–NET aggregates; elevated thrombin generation
MITIGATION DNase treatment in vitro; anti-histone antibodies; anticoagulation; anti-inflammatory therapy
P7 Platelet microparticle seeding
TIMEFRAME minutes–hours
INITIATING EVENT platelet activation at injection site or systemically
MECHANISTIC STEPS Activated platelets shed microparticles exposing phosphatidylserine that nucleate thrombin and bind fibrinogen; microparticles circulate and seed distal clotting
KEY DRIVERS Platelet activation; shear stress; thrombin; exposed PS
DETECTABLE MARKERS Elevated circulating platelet microparticles; annexin V+ particles; increased procoagulant activity
MITIGATION Antiplatelet agents; anticoagulation; reduce platelet activation triggers
P8 Extracellular vesicle (EV) transport of Spike
TIMEFRAME hours–days
INITIATING EVENT cells package Spike into EVs and release them
MECHANISTIC STEPS Spike-bearing EVs circulate and present multivalent Spike on vesicle surface, binding polyanions and recruiting coagulation factors
KEY DRIVERS EV biogenesis; membrane display of Spike; opsonization
DETECTABLE MARKERS Spike detected in EV fractions; nanoparticle tracking; EV proteomics showing Spike and coagulation factors
MITIGATION EV isolation and characterization; immunocapture; anticoagulation if procoagulant
P9 Complement-opsonized aggregate formation
TIMEFRAME hours–days
INITIATING EVENT Spike or Spike complexes activate complement
MECHANISTIC STEPS Complement deposition opsonizes complexes, promotes endothelial activation and leukocyte recruitment, amplifying coagulation via tissue factor
KEY DRIVERS Classical/alternative complement activation; C3b/C5a generation
DETECTABLE MARKERS Elevated C3a/C5a; complement fragments on complexes; endothelial activation markers
MITIGATION Complement inhibitors; anti-inflammatory therapy; anticoagulation
P10 Protease-mediated fibrin remodeling and resistant fragments
TIMEFRAME hours–days
INITIATING EVENT local protease release (neutrophil elastase, bacterial proteases)
MECHANISTIC STEPS Proteases partially degrade fibrin producing atypical fragments that resist fibrinolysis and seed new clot formation
KEY DRIVERS Protease release; altered plasmin activity; crosslinking by FXIII
DETECTABLE MARKERS Abnormal fibrin degradation products; altered D-dimer patterns; plasmin resistance in vitro
MITIGATION Protease inhibitors; enhance plasmin activity; targeted fibrinolysis
P11 Lipid nanoparticle (LNP)-protein aggregation
TIMEFRAME minutes–hours
INITIATING EVENT LNPs carrying mRNA interact with extracellular proteins
MECHANISTIC STEPS LNPs bind serum proteins including fibrinogen or complement components, forming a protein corona that can be procoagulant or immunogenic
KEY DRIVERS LNP surface chemistry; protein corona composition; charge interactions
DETECTABLE MARKERS Protein corona profiling; co-isolation of LNPs with coagulation factors; complement activation
MITIGATION Modify LNP composition; monitor protein corona; anticoagulation if indicated
P12 Antibody-mediated crosslinking and immune complexes
TIMEFRAME days–weeks
INITIATING EVENT anti-Spike antibodies form after vaccination
MECHANISTIC STEPS Antibodies crosslink Spike or Spike-containing fragments creating large immune complexes that activate complement and FcγR on platelets/leukocytes
KEY DRIVERS Timing of seroconversion; antibody isotype and affinity; Fc receptor engagement
DETECTABLE MARKERS Circulating immune complexes; complement activation; platelet activation assays
MITIGATION Immunomodulation (IVIG); avoid heparin if PF4 involvement suspected; non-heparin anticoagulants
P13 Bacterial translocation or endotoxin-triggered coagulation
TIMEFRAME hours–days
INITIATING EVENT local tissue disruption allows bacterial products into circulation
MECHANISTIC STEPS LPS and other PAMPs trigger systemic inflammation, endothelial activation, tissue factor expression and disseminated coagulation
KEY DRIVERS Gut or tissue barrier breach; innate immune activation; TLR signaling
DETECTABLE MARKERS Elevated LPS; cytokines (IL-6); tissue factor; D-dimer
MITIGATION Treat infection; anti-inflammatory therapy; supportive anticoagulation
P14 Oxidative stress–induced endothelial damage
TIMEFRAME hours–days
INITIATING EVENT local oxidative burst or systemic ROS increase
MECHANISTIC STEPS ROS damage endothelial glycocalyx and cells exposing subendothelial procoagulant surfaces and releasing vWF and TF
KEY DRIVERS Oxidative stress; reduced NO; glycocalyx shedding
DETECTABLE MARKERS Elevated soluble thrombomodulin; vWF; markers of oxidative stress
MITIGATION Antioxidants experimentally; endothelial protective strategies; anticoagulation
P15 Protein misfolding,heterologous amyloid seeding (non-fibrin)
TIMEFRAME days
INITIATING EVENT Spike or vaccine components seed misfolding of host proteins
MECHANISTIC STEPS Misfolded proteins form amyloid-like aggregates that bind coagulation factors or platelets, altering hemostasis
KEY DRIVERS Amyloidogenic sequences; molecular chaperone overload; proteostasis stress
DETECTABLE MARKERS Amyloid staining of plasma particulates; protease-resistant aggregates; altered clot structure
MITIGATION Chaperone modulators experimentally; enhance clearance pathways; anticoagulation if prothrombotic
P16 Metal-mediated precipitation, mixed inorganic–protein particles
TIMEFRAME minutes–hours
INITIATING EVENT local release or presence of polyvalent metals (Fe, Ca, Mn)
MECHANISTIC STEPS Metals bridge polyanions and proteins forming mixed inorganic–protein precipitates that trap coagulation factors and cells
KEY DRIVERS Metal availability; pH; polyanion concentration
DETECTABLE MARKERS Particulate metal content by ICP-MS; co-precipitation of coagulation proteins; microscopy
MITIGATION Chelation in vitro; control metal exposure; anticoagulation
Come together or be destroyed.
Introduction: I write this as an educator and researcher who has spent years observing how large systems fracture human bonds while presenting themselves as benevolent guardians. When those systems fail—when they harm rather than heal—the damage is not merely statistical. It is personal, intimate, and enduring. Lives are lost, families are shattered, and grief is often driven underground by silence, ridicule, or fear. What follows is an educational presentation rooted in a simple but profound idea: that ordinary people, acting locally and peacefully, can create meaning, solidarity, and resistance through ritual. This essay proposes a recurring, silent act of remembrance for those believed to have died as a result of the mRNA injections, with good reason and explores why such a ritual matters—not only for mourning, but for community formation, moral clarity, and future resilience.
https://open.substack.com/pub/soberchristiangentlemanpodcast/p/essay-finding-each-other-in-the-darkness
It's like talking to your cat or dog about the mRNA vax/jab they know you care and only mean them well esp at feeding time,
Only our Pets have an excuse = they're animals:
ALL this info I sent revealing every immune boosting food, supplement, herb & drink for optimum health as soon as Covid was announced was coming in Fed., 2020- and I never get a cold or flu,
I then explained all these helps WITH dozensof links so they could validate the scientific research = all so they won't need to get the mRNA Vax when it comes out in 2021.
Sent out to these folks every fact including that *every test animal died given that Vax, and that it was NOT properly vetted after that but WAS just allowed by the CDC+FDA,
And that POTUS Trump was lied to about its effectiveness and reshared that Trump also 1st gave *all the natural immune boosters sent to him by the great Dr. Zelenko,
And also that Fauci was a lying monster about AIDS medicine and 10's of thousands died because of it, and theorphaned black children in NYC he experimented on and killed,
Andthe many Beagle dogs he tortured to death cutting their voice boxs so not to hear the screams,
Still even now most of these same people I reached out to all along do not seem to feel there's a problem,
I have sent this Tom Haviland article by Jim Roguski here to them all already twiceand it'll lijely be ignored:
The viscosity data here is especially compelling because its such a measurable physical property. Going from 30% to 40% grape jelly clots while also seeing these new fibrous structures isn't just anecdotal - embalmers are basically running an unintentional longitudinal study with huge sample sizes. The fact that this pattern held steady across three years of surveys (27-30% prevalence) suggests whatever mechanism is causing this reached some kind of equilibrium. The silence from regulatory agencies tells you everythingyou need to know about institutional priorities.
Yes they know its a GENOCIDE and that the Dolts are complicit...!
THAT is a great question that is important if the Funeral Home tells the family's about the clots in their dead spouse or relative,
Tho be better to show them the clots from their loved one and give the them the glass jar of those clots to take home - huh...!
Do the coroners and medical examiners tell the family of the deceased person that they found these clots in the body?
Almost never. They do not want to upset family members who are already grieving over the loss of a loved one.
However, by staying silent about the clots, the funeral director/embalmer is actually helping Big Pharma get away with murder.
This is awful. No one is being told the truth. Did the deceased die from the clots, or from something else, and are the clots being found during embalming?
Almost always the death certificate just says stroke or "myocardial infarction" (i.e., heart attack) or generic "thrombosis" without describing the TYPE of clot that caused the stroke or heart attack. Very disingenuous if you ask me. These death certificates should be much more specific. I think that they are hiding the fact that many of these deaths are being caused by the WHITE FIBROUS CLOTS on purpose.
It is not Big Pharma, Tom.
THAT is a great question that is important if the Funeral Home tells the family's about the clots in their dead spouse or relative,
Tho be better to show them the clots from their loved one and give the them the glass jar of those clots to take home - huh...!
Thank you so much for interviewing Tom Haviland. More people should be made aware of these clots. I follow Laura and Tom on Substack, after I heard her speak on a local radio show a year ago.
So far I have greatly appreciated these interviews but am unable to open this one at all. Age verification required, the new censorship
Being OBE (Over Bloody Eighty) I will simply be left behind as I will not submit. Free speech or nothing.
This articale shows the blood clots = this all is totally discusting and Genocidal monstrous scheme to cover up and hide from all of ~
"WE the People" = they're all complicit to mass murder and worse a mass coverup while each pockets huge amounts of hush money...!
They all deserve the gallows and IS what they fear for knowing its true...!
As a Christian why do you have them sware to speak truth it's against the one your swearing upon. Yes is to be yes and no to be no. It against the one your swearing upon. Wrong is wrong.
That was an excellent interview which covered all the important points.
I have repeatedly written to my government (in the UK) and had no responses - from my MP or from individual government departments. Letters sent registered mail are not recorded as received. Apparently all mail into the UK government is first "screened" by security so I cannot even know whether this correspondence reached the right desk.
Discussion or questioning of the covid injections is clearly a classified security matter.
Send it return request to be Signed for...!
It was a "signed for" letter - after no response from e-mails.
It was registered mail and tracked OK in the post until it got to Westminster - then nothing!
Registered mail should get a signature to prove receipt. Clearly discussion of the pandemic or the injections goes to security and is classified, so they will not sign or acknowledge receipt..
No doubt a consiracy setup to dis-acknowledge anything they must answer for or to...!
VERY SICK SAD & CROOKED
Thank you James for discussing the embalmer clots . The actual embalmers and their assistants need to be aware of the survey, not just the directors. This topic needs to go viral on X and Instagram and other media platforms. We the People must get the word out . Thanks again to Tom and Laura for their efforts to tell the truth . Repeal the Prep act too . 🙏
This substack site follows the statistical drop in births since 2020, and the elevated excess death rates.
I recommend this site her work is excellent. She has some very useful insights.
https://supersally.substack.com/
Depopulation at its finest 😞😢
"Sally" noticed that the death rates in Pilipino CHILDREN rose when the ADULT vaccines rolled out. Shedding?
The fertility rates have dropped substantially ion all countries she follows. It may be that some of the statistical effects suggest targeting of certain populations for specific results, but it is too early yet to know, and preexisting population trends complicate the picture.
These interviews are a treasure trove of truth!
Can't say this often enough! KEEP FIGHTING! All the perps who pushed this greatest crime against humanity, all the way down to the local level, must get their comeuppances!
MISTAKES! WERE NOT MADE!
It was NEVER about health! The Powers That Should Not Be were ALWAYS about they want you DEAD or a SLAVE! This is a painful truth to accept but we the people must wake up and fight back! And toxic injections/pills were/are a huge part of their arsenal!
Can't say this often enough! The Military/Industrial Complex and the Biowarfare/industrial Complex, WEF agenda and the evils assaulting humanity are from one and the same source - it is the 99% against the diabolical GREED of the 0.01% who should not be in charge of anything!
The monsters in human skin suits who rule the world get a sadistic vampiric thrill and boost from perpetrating the vilest most demonic crimes against the most vulnerable (babies and small children) and then corrupting the system to get away with it scot free! We the People must stop them, there are a lot more of us than them!
Please check out this substack! ponerology.substack.com
And BIG pHARMa is an arsenal making permanently sickly addicted slaves dependent on their products - the complete opposite of actual health.
Peddling pure poison! Folks have to wake up to reality: health comes from organic diet, daily exercise and clean living and never from a needle or a pill except in dire, rare traumatic injuries.
Can't say this often enough!
SCREW THE HYPOCHONDRIA GERMAPHOBIC FEAR HYSTERIA! DO NOT CONSENT! Avian flu is for the birds! RESIST!
Proudly ANTI-VAXX! Reiterating for the sake of newbies and to support this post.
Ban all vaccine jabs! There has never been a 'safe and effective' vaccine since Edward Jenner's fraud over 200 years ago as per 'Dissolving Illusions' by Suzanne Humphries and 'Turtles All the Way Down' by Anonymous. Health can never come from a needle or pills, but from healthy eating, healthy exercise and healthy living! virustruth.net
JAB INJURIES: GROSS CALAMARI BLOOD CLOTS/AUTISM TSUNAMI/SADS/TURBO CANCER/BIZARRE TERMINAL ILLNESSES: More tragic victims of the ruling parasite genocidal enslavement agenda, sacrificed on the altar of psychopathic greed and hatred of humanity.
Divide and rule! Agents provocateurs anyone, FALSE FLAGS, propaganda social engineering psyops? Keeping us proles at each others' throats while the globalist technocrat predators laugh all the way to the BIS and The Bank of Rothschild's!
BURN BACK BETTER!
HELL NO TO GENESIS MISSION/STARGATE/DEEPSEEK! HELL NO TO AI! technocracy.news
Life everywhere is being assaulted by THE TECHNOCRATIC OMNIWAR! RESIST! DO NOT CONSENT TO ALL THINGS DIGITAL, 'SMART', AI, 5G, NO CASH - ALL OF IT! dhughes.substack.com Technocrat ruling class psychos get a sadistic thrill from their powers over life and death and hurting all who stand in their way and they need the resources worldwide to build their digital total slavery control grids (herd survivors into 15 minute city digital prisons)!
AI is designed to be anti-human/anti-life programmed by technocrat control freak psychos - garbage in = garbage out. Everyone got along just fine without all these absurd and downright satanic electronic gadgets that did not exist until recently. NOBODY NEEDS THIS AI CRAP!
PSYCHOPATHS! MEGALOMANIACS!
This horrifying Congress Critters, Gates, Governor 'Gruesome Newscum', 'Lone Scum', Soros, 'Benedict' Biden and Harris and even Trump, Vance, and 'Ramaswampy' et al are blatant fully owned and operated puppets of their globalist technocrat parasite masters same as other numerous 'PUBLIC SERPENTS' infesting by design from above, the bureaucratic apparatus.
CREATIVITY! ARTISTRY! IMAGINATION! SPIRITUALITY! HUMOR! LOVING KINDNESS! These are the best ways to fight THEM!
Bless and thank you for doing what you do.
I was at emergency the other day in Pembroke, On. Canada and the triage nurse adk if I had
the flu and covid jabs I told her no to both and further said I would not ever take an MRNA jab! I was shocked they are still pushing this not safe and not effective jab! I believe the flu
shot is now MRNA! Last flu shot 2019 gave me a slight stroke 5 days after! I now do not
take any jabs!! FT!
Great, important interview. James, your NotSafeandNotEffective website is the best and most thorough compilation of covid shot information I have seen, so I share it as much as possible. Thank you for this greate site.
Blessings and appreciation from Sydney Australia.
So much death n destruction to come to terms with. Even more demanding- to come to terms with , the complicity of our governments and medical professionals .